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14.08.2012 КТ Ангио Сонных Артерий. Врожденная гипоплазия внутренней сонной артерии

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 Ага, красиво, так понимаю по

 Ага, красиво, так понимаю по стостоянию сосудов и костей шо стеноз врожденный с нормальной компенсацией за счет позвоночных артерий?

 
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+1

+1

 
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Врожденная гипоплазия

Врожденная гипоплазия внутренней сонной артерии.

 

 

 Congenital Hypoplasia of the Internal Carotid Artery
Topic 9436 - Created: 2010-01-03 13:56:08-05 - Modified: 2010-01-06 15:04:19.090909-05
ACR Index: 1722.142

Congenital absence or hypoplasia of one or both internal carotid arteries (ICAs) is a rare developmental abnormality usually discovered incidentally by imaging done for other reasons. Patients are generally asymptomatic due to well developed collateral circulation Reported incidence is .01%. Internal carotid dysgenesis can be classified as agenesis (no carotid canal or vascular remnant), aplasia (vascular remnant and hypoplastic carotid canal), or hypoplasia (small caliber, patent lumen). The diagnosis is favored when CT imaging of the skull base demonstrates a hypoplastic carotid canal and the ipsilateral common carotid artery is asymmetrically narrow in caliber.

Etiology is unkown. The internal carotid artery is thought to develop from segments of seven different embyrologic vessels. Maldevelopment of one of these segments may lead to poor development of the internal carotid artery. The artery forms in the fourth embryologic week followed soon after by the bony carotid canal. The external carotid artery is rarely absent in dysgenesis of the internal carotid, likely reflecting a different embryologic origin.

   The ipsilateral ICA branches can be supplied from the Circle of Willis, branches of the external carotid, persistent embryonic arteries, or intercavernous anastamoses. One classification system for collateral flow is as follows:

A: Ipsilateral ACA fills via the ACoA; ipsilateral MCA via the PCoA
B: ACA and MCA fill via the ACoA
C: Both ICAs are absent and both ACAs and MCAs fill via persistent vascular structures originating from the basilar system
D: Aplastic cervical ICA; supraclinoid ICA fills via intercavernous collateral from contralateral ICA
E: Bilateral hypoplastic ICAs- give rise only to ACAs
F: Bilateral aplastic cervical ICAs; precavernous ICAs filled by ECA branches in skull base

The supplying arteries are at higher risk of aneurysm formation and associated complications, and should be closely evaluated on non-invasive imaging studies. A diagnostic angiogram may be indicated to clarify indeterminate findings. Incidence of intracranial aneurysms may be as high as 34% compared to 3-4% in the general population, likely due to increased hemodynamic pressure in the collateral arteries.

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